Helsinki Academic Medical Center

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Thymic Development and Selection of T Lymphocytes Microbiology, Medical Center, Helsinki

Uutuuskirjoja Terkossa 02 / 2014

Lastenneurologia / toimittajat: Helena Pihko, Leena Haataja, Heikki Rantala ”Kirjassa esitellään lapsen normaali kehitys ja sen yleisimmät poikkeavuudet, kuten puheen- ja kielenkehityksen sekä motoriikan häiriöt, oppimisvaikeudet, aktiivisuuden ja tarkkaavuuden häiriö, autismikirjon häiriöt ja kehitysvammaisuus. Lastenneurologia kuvaa tavallisia lasten neurologisia oireita ja sairauksia, kuten päänsärky, sekä harvinaisia neurologisia sairauksia, kuten neuromuskulaarisairaudet. Mukana ovat…

Molecular Mechanisms in Legionella Pathogenesis: 376 (Current Topics in Microbiology and Immunology) Agricultural Science, Microbiology, Medical Center, Textbook, Helsinki, Kindle, Ebooks, Products, Class Books

Uutuuskirjoja Terkossa 02 / 2014

Lastenneurologia / toimittajat: Helena Pihko, Leena Haataja, Heikki Rantala ”Kirjassa esitellään lapsen normaali kehitys ja sen yleisimmät poikkeavuudet, kuten puheen- ja kielenkehityksen sekä motoriikan häiriöt, oppimisvaikeudet, aktiivisuuden ja tarkkaavuuden häiriö, autismikirjon häiriöt ja kehitysvammaisuus. Lastenneurologia kuvaa tavallisia lasten neurologisia oireita ja sairauksia, kuten päänsärky, sekä harvinaisia neurologisia sairauksia, kuten neuromuskulaarisairaudet. Mukana ovat…

Pathogenesis of Mycobacterium Tuberculosis and Its Interaction With the Host Organism Microbiology, Medical Center, Helsinki

Uutuuskirjoja Terkossa 02 / 2014

Lastenneurologia / toimittajat: Helena Pihko, Leena Haataja, Heikki Rantala ”Kirjassa esitellään lapsen normaali kehitys ja sen yleisimmät poikkeavuudet, kuten puheen- ja kielenkehityksen sekä motoriikan häiriöt, oppimisvaikeudet, aktiivisuuden ja tarkkaavuuden häiriö, autismikirjon häiriöt ja kehitysvammaisuus. Lastenneurologia kuvaa tavallisia lasten neurologisia oireita ja sairauksia, kuten päänsärky, sekä harvinaisia neurologisia sairauksia, kuten neuromuskulaarisairaudet. Mukana ovat…

One Health - the Human-animal-environment Interfaces in Emerging Infectious Diseases: The Concept and Examples of. First Health, Microbiology, Medical Center, Helsinki

Uutuuskirjoja Terkossa 02 / 2014

Lastenneurologia / toimittajat: Helena Pihko, Leena Haataja, Heikki Rantala ”Kirjassa esitellään lapsen normaali kehitys ja sen yleisimmät poikkeavuudet, kuten puheen- ja kielenkehityksen sekä motoriikan häiriöt, oppimisvaikeudet, aktiivisuuden ja tarkkaavuuden häiriö, autismikirjon häiriöt ja kehitysvammaisuus. Lastenneurologia kuvaa tavallisia lasten neurologisia oireita ja sairauksia, kuten päänsärky, sekä harvinaisia neurologisia sairauksia, kuten neuromuskulaarisairaudet. Mukana ovat…

Uutuuskirjoja Terkossa 02 / 2014 – Meilahden kampuskirjasto Terkko - blogi Medical Center, Helsinki

Uutuuskirjoja Terkossa 02 / 2014

Lastenneurologia / toimittajat: Helena Pihko, Leena Haataja, Heikki Rantala ”Kirjassa esitellään lapsen normaali kehitys ja sen yleisimmät poikkeavuudet, kuten puheen- ja kielenkehityksen sekä motoriikan häiriöt, oppimisvaikeudet, aktiivisuuden ja tarkkaavuuden häiriö, autismikirjon häiriöt ja kehitysvammaisuus. Lastenneurologia kuvaa tavallisia lasten neurologisia oireita ja sairauksia, kuten päänsärky, sekä harvinaisia neurologisia sairauksia, kuten neuromuskulaarisairaudet. Mukana ovat…

PLOS Genetics: Re-sequencing Expands Our Understanding of the Phenotypic Impact of Variants at GWAS Loci Genetic Variation, Human Genome, Cardiovascular Disease, Medical Center, Helsinki, Disorders, Heart Disease

Re-sequencing Expands Our Understanding of the Phenotypic Impact of Variants at GWAS Loci

Author Summary Abnormal serum levels of various metabolites, including measures relevant to cholesterol, other fats, and sugars, are known to be risk factors for cardiovascular disease and type 2 diabetes. Identification of the genes that play a role in generating such abnormalities could advance the development of new treatment and prevention strategies for these disorders. Investigations of common genetic variants carried out in large sets of research subjects have successfully pinpointed…

PLOS ONE: Prenatal and Childhood Growth, and Hospitalization for Alcohol Use Disorders in Adulthood: The Helsinki Birth Cohort Study Non Communicable Disease, Cohort Study, Medical Center, Helsinki, Disorders, Birth, Childhood, Alcohol, Rubbing Alcohol

Prenatal and Childhood Growth, and Hospitalization for Alcohol Use Disorders in Adulthood: The Helsinki Birth Cohort Study

Background Small birth size - an indicator of a sub-optimal prenatal environment - and variation in growth after birth have been associated with non-communicable diseases in later life. We tested whether birth size or growth in childhood associated with the risk of hospital admission for alcohol use disorders (AUDs) from early to late adulthood. Methods The sample comprised 6544 men and 6050 women born between 1934 and 1944 in Helsinki, Finland. Data on anthropometric measures were extracted…

Rapid Molecular Characterization of Acinetobacter baumannii Clones with rep-PCR and Evaluation of Carbapenemase Genes by New Multiplex PCR in Hospital District of Helsinki and Uusimaa Study Materials, Medical Center, Helsinki

Rapid Molecular Characterization of Acinetobacter baumannii Clones with rep-PCR and Evaluation of Carbapenemase Genes by New Multiplex PCR in Hospital District of Helsinki and Uusimaa

Multidrug-resistant Acinetobacter baumannii (MDRAB) is an increasing problem worldwide. Prevalence of carbapenem resistance in Acinetobacter spp. due to acquired carbapenemase genes is not known in Finland. The purpose of this study was to examine prevalence and clonal spread of multiresistant A. baumannii group species, and their carbapenemase genes. A total of 55 Acinetobacter isolates were evaluated with repetitive PCR (DiversiLab) to analyse clonality of isolates, in conjunction with…

PLOS ONE: and Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cell. Cells Activity, Gene Expression, Thing 1, Transcription, Medical Center, Helsinki

FOXL2, GATA4, and SMAD3 Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cells

Aberrant ovarian granulosa cell proliferation and apoptosis may lead to granulosa cell tumors (GCT), the pathogenesis of which involves transcription factors GATA4, FOXL2, and SMAD3. FOXL2 gene harbors a point mutation (C134W) in a vast majority of GCTs. GATA4 is abundantly expressed in GCTs and its expression correlates with poor prognosis. The TGF-β mediator SMAD3 promotes GCT cell survival through NF-κB activation, and interacts with FOXL2. Here, we find that the expression patterns of…

PLOS ONE: and Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cell. Gene Expression, Transcription, Medical Center, Helsinki, Positivity, Activities, Optimism

FOXL2, GATA4, and SMAD3 Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cells

Aberrant ovarian granulosa cell proliferation and apoptosis may lead to granulosa cell tumors (GCT), the pathogenesis of which involves transcription factors GATA4, FOXL2, and SMAD3. FOXL2 gene harbors a point mutation (C134W) in a vast majority of GCTs. GATA4 is abundantly expressed in GCTs and its expression correlates with poor prognosis. The TGF-β mediator SMAD3 promotes GCT cell survival through NF-κB activation, and interacts with FOXL2. Here, we find that the expression patterns of…

PLOS ONE: and Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cell. Gene Expression, Transcription, Medical Center, Helsinki, Positivity, Optimism

FOXL2, GATA4, and SMAD3 Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cells

Aberrant ovarian granulosa cell proliferation and apoptosis may lead to granulosa cell tumors (GCT), the pathogenesis of which involves transcription factors GATA4, FOXL2, and SMAD3. FOXL2 gene harbors a point mutation (C134W) in a vast majority of GCTs. GATA4 is abundantly expressed in GCTs and its expression correlates with poor prognosis. The TGF-β mediator SMAD3 promotes GCT cell survival through NF-κB activation, and interacts with FOXL2. Here, we find that the expression patterns of…

PLOS ONE: and Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cell. P Value, Gene Expression, Transcription, Medical Center, Helsinki, Positivity, Activities, Optimism

FOXL2, GATA4, and SMAD3 Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cells

Aberrant ovarian granulosa cell proliferation and apoptosis may lead to granulosa cell tumors (GCT), the pathogenesis of which involves transcription factors GATA4, FOXL2, and SMAD3. FOXL2 gene harbors a point mutation (C134W) in a vast majority of GCTs. GATA4 is abundantly expressed in GCTs and its expression correlates with poor prognosis. The TGF-β mediator SMAD3 promotes GCT cell survival through NF-κB activation, and interacts with FOXL2. Here, we find that the expression patterns of…

PLOS ONE: and Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cell. Gene Expression, Transcription, Medical Center, Helsinki, Positivity, Optimism

FOXL2, GATA4, and SMAD3 Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cells

Aberrant ovarian granulosa cell proliferation and apoptosis may lead to granulosa cell tumors (GCT), the pathogenesis of which involves transcription factors GATA4, FOXL2, and SMAD3. FOXL2 gene harbors a point mutation (C134W) in a vast majority of GCTs. GATA4 is abundantly expressed in GCTs and its expression correlates with poor prognosis. The TGF-β mediator SMAD3 promotes GCT cell survival through NF-κB activation, and interacts with FOXL2. Here, we find that the expression patterns of…

PLOS ONE: and Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cell. Gene Expression, Medical Center, Helsinki, Pattern, Patterns, Model, Swatch

FOXL2, GATA4, and SMAD3 Co-Operatively Modulate Gene Expression, Cell Viability and Apoptosis in Ovarian Granulosa Cell Tumor Cells

Aberrant ovarian granulosa cell proliferation and apoptosis may lead to granulosa cell tumors (GCT), the pathogenesis of which involves transcription factors GATA4, FOXL2, and SMAD3. FOXL2 gene harbors a point mutation (C134W) in a vast majority of GCTs. GATA4 is abundantly expressed in GCTs and its expression correlates with poor prognosis. The TGF-β mediator SMAD3 promotes GCT cell survival through NF-κB activation, and interacts with FOXL2. Here, we find that the expression patterns of…

PLOS ONE: A “Candidate-Interactome” Aggregate Analysis of Genome-Wide Association Data in Multiple Sclerosis Jc Virus, Open Reading Frame, P Value, Multiple Sclerosis, Medical Center, Helsinki

A “Candidate-Interactome” Aggregate Analysis of Genome-Wide Association Data in Multiple Sclerosis

Though difficult, the study of gene-environment interactions in multifactorial diseases is crucial for interpreting the relevance of non-heritable factors and prevents from overlooking genetic associations with small but measurable effects. We propose a “candidate interactome” (i.e. a group of genes whose products are known to physically interact with environmental factors that may be relevant for disease pathogenesis) analysis of genome-wide association data in multiple sclerosis. We looked…

PLOS ONE: A “Candidate-Interactome” Aggregate Analysis of Genome-Wide Association Data in Multiple Sclerosis Jc Virus, Open Reading Frame, P Value, Multiple Sclerosis, Medical Center, Helsinki

A “Candidate-Interactome” Aggregate Analysis of Genome-Wide Association Data in Multiple Sclerosis

Though difficult, the study of gene-environment interactions in multifactorial diseases is crucial for interpreting the relevance of non-heritable factors and prevents from overlooking genetic associations with small but measurable effects. We propose a “candidate interactome” (i.e. a group of genes whose products are known to physically interact with environmental factors that may be relevant for disease pathogenesis) analysis of genome-wide association data in multiple sclerosis. We looked…